The search space is optimized i sample count. We will most likely find solutions on the virus. From a coloring perspective, we are adding energy to force slightly more coorting pairs than the surfcae can support.
We are not relativistic, we can alter the search strings and relative concentrations. The extra freedom allows us to get the two -ilog(i) much closer. The ratio is both search time and concentration. So we have the 1/fifth and a 1/seventh error update, in counts. Setting N sets the last moment and we should be hitting one out of four searches. Every four steps across the vrus surface,there should be a match, if the virus surface is reasonably large.
Our ability to set N is limited. The channel is flexible but wears oou when white cell congestion is out of balance.
The optimum consumption leaves the maximum distribution of cytokines recycled through the lymph, reinforcing the successful pairs concentration., There is almost certainy a two sided chain identifier already.
The official model follows this schematic:
How to generate antibodies
Yes, reading through it has the analogs. It has a Heavy chain and a light chain search list, a parit for each polyclonal B-Cell.
The antibody is exogenous digestion by the B-cell, it seems, a braking down of the food to digestable protein chains. This happens in he differentiation phase.
There is a two step process, sniff and reject or sniff and digest. That is an energy consuming none redundancy minimization. It give the channel much more adaptive flexibility, but make is unstable. The Heayy chain search B-cell gets an extra sample, relative to the light chain.
The extra look lays in that half the time there is no HC match and status quo ante prevails., except the rejecting B-cell moves away from the target. This is the relativity gain. Basiclly the system forces the closed virus to obey a flat surface, and it tears apart, cannot maintain the density.
he best vaccine gets the HC chain immunoglobulin right, then lets the natural digestion handle the light chains. Do not bother with macrophage, yet. (Some day we will want ot kill them).
Basically these B-Cells have five major food groups is one way to think of it, there has been much path merging in the error update tree. Then thee are a large number of preferences for arrangements of these food groups in a well balanced meal. The second look means the HC B-cell will alweays have two updaes pending in the queue, when there was one previously. But there are basically five odels of the HC B cells floating around. It still rolls like a five counter. That collapse in the update tree means takes fewer paths to identify the next best button to collor on the ballon, but more kinetic energy supplied by the docs.
Handling event probability with the second look is to ignore time on the rejects, as if they did not happen. We end up making the sevens counter a three counter, with partial loss. This becomes z,3,5 and one out of 15 paths will keep the amount of rendundancy we specify. In other words, we will at time take out a chunk of virus and lose nad sometimes take out a chunk of good stuff and win. Z will be about two. We will also be losing most of what the macrophage is eating. But en of those misses will do not harm except eat energy. The virus surface is large, we will get the hit we need to decompose it.
It is when we set the concentration of HC B cells relative to LC B cells. That concentration favors the maximum paths on the virus surface. The ratio that maximizes he number of matches suitable for dissolution, within our damage range. We let relativity do the rest, partition the HC B cells. The HC cells dwell that one extra step when they much a long peptide on the surface. They will tend to disperse along the surface and LC B cells maximally fill in the colors relative to any other concentration.
Adding energy and getting the second look opens up wide possibilities.
After immunization, blood IgM levels increase first.With repeated immunization, IgG levels increase.Changing the concentration of B-cells according to their search list. An immunoglobulin is a search list. A patjhogen in polycloanl is dientified by the intersection of two classes. Basically the way the math predicts.
The antibody is exogenous digestion by the B-cell, it seems, a braking down of the food to digestable protein chains. This happens in he differentiation phase.
Chemokinesis is chemically prompted kinesis, a motile response of unicellular prokaryotic or eukaryotic organisms to chemicals that cause the cell to make some kind of change in their migratory/swimming behaviour.
There is a two step process, sniff and reject or sniff and digest. That is an energy consuming none redundancy minimization. It give the channel much more adaptive flexibility, but make is unstable. The Heayy chain search B-cell gets an extra sample, relative to the light chain.
The extra look lays in that half the time there is no HC match and status quo ante prevails., except the rejecting B-cell moves away from the target. This is the relativity gain. Basiclly the system forces the closed virus to obey a flat surface, and it tears apart, cannot maintain the density.
he best vaccine gets the HC chain immunoglobulin right, then lets the natural digestion handle the light chains. Do not bother with macrophage, yet. (Some day we will want ot kill them).
Basically these B-Cells have five major food groups is one way to think of it, there has been much path merging in the error update tree. Then thee are a large number of preferences for arrangements of these food groups in a well balanced meal. The second look means the HC B-cell will alweays have two updaes pending in the queue, when there was one previously. But there are basically five odels of the HC B cells floating around. It still rolls like a five counter. That collapse in the update tree means takes fewer paths to identify the next best button to collor on the ballon, but more kinetic energy supplied by the docs.
Handling event probability with the second look is to ignore time on the rejects, as if they did not happen. We end up making the sevens counter a three counter, with partial loss. This becomes z,3,5 and one out of 15 paths will keep the amount of rendundancy we specify. In other words, we will at time take out a chunk of virus and lose nad sometimes take out a chunk of good stuff and win. Z will be about two. We will also be losing most of what the macrophage is eating. But en of those misses will do not harm except eat energy. The virus surface is large, we will get the hit we need to decompose it.
It is when we set the concentration of HC B cells relative to LC B cells. That concentration favors the maximum paths on the virus surface. The ratio that maximizes he number of matches suitable for dissolution, within our damage range. We let relativity do the rest, partition the HC B cells. The HC cells dwell that one extra step when they much a long peptide on the surface. They will tend to disperse along the surface and LC B cells maximally fill in the colors relative to any other concentration.
Adding energy and getting the second look opens up wide possibilities.
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