So we make predictions about the white cell partition

It must break into groups 4,5,7; and an antibody is really an adjustment in the ration of five and seven. The anti body cloning is via a math in the five and seven path identifiers. The control is likely in the lymphc system. At he lymph gate ways the antigen encode by molecular ratio. In the medium, ratios are optimum for binding on some likely target, beating it at the game of chinese remainder. We need to think that tthe food mix changes, the gateway encoding changes.  Send the antigen in two parts, in the proper ratio to represent the virus surface.

Pick the unique combination of seven and five chains proteins on the virus surface.  Insert the antigens into the system in proportion to their surface ratio. To the extent the virus surface is unique, ti can be targeted. If you measure  current antibody profile,then make the concentration a differential on that.

It should be possible to install the antigens at site specific spots. Virus immunity failure is a result of congested antigen at lymph entries.  The solution for those would be direct antibodies. Erase some part of the menu.

The corona antigens likely have a history of crowding space. A strong targeted antibody for the corona family clears the channel,corona can be retuned and restarted. Making the semi independent paths allows safe immunity. One really doesn't need a pure antigen. Huffman encoding:

The most accepted view is that the TCR engages in kinetic proofreading. The kinetic proofreading model proposes that a signal is not directly produced upon binding but a series of intermediate steps insure a time delay between binding and signal output. Such intermediate "proofreading" steps can be multiple rounds of tyrosine phosphorylation. These steps require energy and therefore do not happen spontaneously, only when the receptor is bound to its ligand. This way only ligands with high affinity that bind the TCR for a long enough time can initiate a signal. All intermediate steps are reversible, such that upon ligand dissociation the receptor reverts to its original unphosphorylated state before a new ligand binds.[

The T cells search in log time through the sequence.  I guess it is the way the sequence is folded that holds the encoding tree. When they say reversible they mean two things, adiabatic, but more, an extra separator lets them take a reverse entropy path, at the cost of energy.  This is actually the relativity effect. The T Cell can search with some variability about the likely target peptide. And this extra signal to another T-Cell is enough to decompose before the virus can respond.  Each T-cell contains its own set of peptide matches, but there will be two groups attacking the typical virus, relatively prime..

But that pause in match delivers a short cytokine exchange with the neighbor on the surface. It is both theories together, the theory on relative concentrations and the theory of a maintained redundancy in error updates.  The white cell get an extra cross step as it colors the virus. it can reverse a move now and then due to the redundant axis taken out by the macrophage, two options present at each roll of the white cell.  Then multipliciy, the ability to increase N, the white cell count over the surface, in proper concentration, increases the likelihood of finding just the unique sequence of two prime peptides  very specific, and their removal then is one step ahead of the virus recomposing..

Over the surface, the white cell searching the shorter chain will roll over faster, and getss more samples, setting more color.   The long chain hunter likely riding on top looking for the hole. That is 35 different paths forward, slightly more with the energy input. But that is one step through the sequence, should kill one virus. I guess the five and seven counters have different folds which expose the markers in log steps.

The system fails when the concentrations cannot be adjusted, the search and match steps increase while virus recomposes in the usual steps. Then the white cells cannot match and they constantly issue cytokines in the search. Meanwhile the macrophage is running away with the hi high rate counter.  Likely the antigen vaccines at the lymph system got congested with yeas of virus selection.

The T-Cell peptide selector was a six element, but switching it down to either five or up to seven gets  relative prime. But there is arbitrage, energy needed.,

To get 23^2 maximum combinations, the five groups searches by five, the seven by seven.   There is path thinning.  But searching 35 paths is a lot of energy.