The whole thing is built on differentiation of white cell

There is one type, finite, from one place. There are a finite number of choices in the next step.  We have our band limited one thing upon which we can understand how to partition. All of this coloring theory applies, an we control N, on the margin. Forget the virus for a moment, think of the limited set pf second helpings the white cells might want. On the margin, we want them a bit more hungry for the composition of the virus surface.  A much simpler task.

Here on this blog, a week ago, I was talking the four color problem, and it matched the DNA explanations I was seeing.  It is really a matter of cutting up that white cell genome into the various factors of the two relative primes. Really be more of a google search engine on that surface, speed up searches by riming search path, limiting commutativity. And watching for congestion in the side.

The fourth color is induced on the proteins being eaten. It becomes a free parameter due to the docs control of total concentration, N. With some danger, the doc has adaptability, the capacity will expand enough under control of the doc and the search times.

Juts yesterday a bunch of pros were solving the three color. Getting it in the limit, and in the finite, and over repeated trials. Three different models, valid under the assumptions. Piece of cake, really sandbox this, done deal.

My guess is that as long as we can solve the virus genome, we can target it optimally, and change targets adaptively.  We should be able to change our concentrationt seasonally, attacking the combination of flu and corona.    But we will encounter the rare virus, the one that needs specific genome cuts with search paths trimmed for the beast.